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The goal isnt to treat a marker, its to understand why the check-engine light is on in the first place

This is simply because our cells make NAD, and as we age our bodies cant replace the cells that die as quickly with new ones

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Collectively, these data again suggest that GIPR antagonism, unlike agonism, mimics GLP-1R agonism in the DVC and that GIPR, unlike GLP-1R, is the primary target for MAR709 in the DVC