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Proteases in the peritoneal fluid appear to degrade GLP-1 faster than normal, creating a pro-inflammatory environment where the natural braking mechanisms on inflammation are weakened

The relationship between the bloodbrain-barrier and the central effects of glucagon-like peptide-1 receptor agonists and sodium-glucose cotransporter-2 inhibitors

Notably, GLP-1 receptor agonists also slow biliary motility, which can contribute to the development of bile ductblocking sludge and stones, resulting in acute pancreatitis

The American Diabetes Association (ADA) and the European Association for the Study of Diabetes (EASD) recommend considering GLP-1R agonists for T2DM patients who do not have established CVD but are at high risk for ASCVD