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Depending on the balance between ROS and the availability of antioxidants in the microenvironment of the cell, antioxidants can inhibit or delay the initiation or propagation of oxidative chain reaction and thus prevent or repair cell damage caused by reactive oxygen [8]

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Preclinical studies have used various neurotoxin-based models to mimic the oxidative stress and dopaminergic neurodegeneration observed in PD

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