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Key documented mechanisms include stimulation of collagen and glycosaminoglycan synthesis at nanomolar concentrations, modulation of matrix metalloproteinase activity, promotion of angiogenesis through copper-dependent endothelial signaling, suppression of NF-kB inflammatory signaling, and modulation of over 4,000 human genes across repair and defense pathways (Pickart and Margolina, International Journal of Molecular Sciences, 2018, DOI: 10.3390/ijms19071987)
In terms of actual lung disease, a possible scenario may be that symptoms, including excessive inflammation and oxidative stress, may be tied to these adaptive responses and may only begin to arise when these adaptive responses fail
Fate of circulating oxalate in rats
Dringen R, Kranich O
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