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[5] The researchers also note that IR and IGF1R act as identical portals to the regulation of gene expression, with differences between insulin and IGF-1 effects due to a modulation of the amplitude of the signal created by the specific ligand-receptor interaction. The IGF-1R is referred to as the physiologic receptor due to its potential higher affinity (approximately 100 times higher) for IGF-1 as compared to the insulin receptor

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Giordano T, Steagall PV, Ferreira TH, Minto BW, de Sa Lorena SE, Brondani J, et al

It helps maintain glutathione in its "reduced" or active state, which is when it is most effective at fighting free radicals