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Keywords: cardiorenal outcomes, chronic kidney disease, diabetes, obesity, nephroprotection Introduction Driven by both traditional and emerging non-traditional risk factors, the prevalence of chronic kidney disease (CKD) has increased significantly in recent years

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In addition to the blood glucose-lowering effect of these anti-hyperglycemic drugs (and thus restricting glucose availability to cancer cells), some of the tested anti-hyperglycemic drugs (e.g., metformin) have an anti-cancer potential via the inhibition of several different cancer-promoting pathways and by the activation of pathways that cause cancer cell death [8,9]

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