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Therefore, this suggests that lead exposure induced oxidative stress in the kidney

Supporting this, H 2 S n compounds have been found to induce calcium mobilization by activating transient receptor potential A1 channels and show a potency that is 320 times greater than that of parental H 2 S (27)

Moreover, acetate and butyrate decrease the levels of reactive oxygen species (ROS), enhance the secretion of anti-inflammatory cytokine IL-10 and immunoregulatory factor IFN-, thereby further mitigating the inflammatory response (68)