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Researchers investigate receptor binding, intracellular signaling, and metabolic pathway responses associated with this concurrent triple activation, comparing it with single- and dual-agonist profiles in controlled laboratory settings [2]

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Research utilizing in vivo models has demonstrated the protective role of endogenous catalase in the early stages of non-alcoholic fatty liver disease by preserving liver redox balance and shielding against hepatic injury induced by high-fat diets [128]