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Furthermore, an imbalance in iron homeostasis has been linked to respiratory diseases, with iron overload and mitochondrial dysfunction playing significant roles in pulmonary dysfunction and pulmonary fibrosis [47]

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(2012) revealed during postmortem examination of GLP intoxication cases that toxic effects were caused by their ability to erode tissues including mucous membranes and linings of the gastrointestinal and respiratory tracts

As such, plasma changes in these acylcarnitines suggest that these effects are likely to be peripheral rather than central manifestations of this illness (Abdullah et al., In mouse models simulating GWS, mice were exposed to pyridostigmine bromide (PB) and permethrin (PER) for 10 days at 12 weeks of age, with proteomic analysis of brain extracts conducted at 5 months post-exposure